Critically, between these extremes lies equi-modal cortex in the inferior temporal and fusiform gyri that responds similarly across modalities and presumably learn more codes transmodal structure. In summary, the process of extracting meaning from our experience with objects involves the fusion of complex sets of information from sensory inputs, motor programmes and verbal experience. We have demonstrated that one key aspect of

this process, the integration of individual features into coherent concepts, depends critically on the ATLs. We are indebted to the patients and their carers for their generous assistance with this study. We thank Prof. Alistair Burns, Prof. Roy Jones and Dr. Roland Zahn for referring patients to us. The research was supported by an MRC Programme Grant (MR/J004146/1), an NIHR Senior Investigator Award to M.A.L.R., an MMHSCT Stepping Stone Award to P.H. and a Wellcome Trust Institutional Strategic Support Fund (ISSF) award (097820) to the University of Manchester. “
“Face recognition is an important cognitive skill that most people take for granted, yet it depends on a complex set of cognitive and neural processes (Bruce and Young, 1986 and Haxby et al., 2000). In some individuals this process can be selectively disrupted, resulting in a condition termed “prosopagnosia” or “face-blindness”.

While prosopagnosia can be acquired following brain injury (e.g., Damasio, Damasio, & Van Hoesen, 1982), many Dabrafenib datasheet more individuals simply fail to develop normal face recognition Liothyronine Sodium abilities (e.g., Bate et al., 2009, Bate et al., 2008 and Behrmann and Avidan, 2005; Bentin, Deouell, & Soroker, 1999; Duchaine et al., 2007, Duchaine and

Nakayama, 2006, Jones and Tranel, 2001 and Schmalzl et al., 2008). The latter form of the disorder has been termed ‘developmental prosopagnosia’ (DP; but for a discussion of terminology see Susilo & Duchaine, 2013), and has been attributed to a failure to develop the visual recognition mechanisms necessary for successful face recognition, despite intact low-level visual and intellectual functions. Interestingly, there also appears to be a genetic component to the disorder in at least some individuals (Duchaine et al., 2007 and Grueter et al., 2007). In the last decade it has become increasingly clear that DP represents a significant clinical disorder, with recent reports suggesting that two percent of the population have the condition (Bowles et al., 2009 and Kennerknecht et al., 2006). Although many studies have investigated the cognitive, neural and genetic basis of DP, little attention has been directed towards improving face recognition in these individuals. While some researchers have attempted to remedy face processing deficits using extensive visual training programmes (e.g., DeGutis et al., 2007 and Schmalzl et al., 2008), recent evidence suggests that an alternative methodology warrants investigation.